The results revealed that Kanserin could reversed the depressed marrow to some extent in the malignant tumor patient following chemotherapy. 结果显示康赛宁能拮抗化疗对恶性肿瘤病人的骨髓抑制作用。
Because the patient had depressed marrow induced by occupational disease and polycystic kidney, So the postoperative immunosuppressive therapy was interfered, that the patient oc-cured two rejection, but they were controled rapidly by the treatment of large dosage methy prednisolone. 术后因患者职业病所致骨髓抑制和多囊肾及肾功能减退影响了免疫抑制剂的应用,曾发生二次排斥反应,经MP冲击治疗很快控制。
Conclusion: rhG-CSF has the potential to accelerate the recovery of depressed marrow and granulocytopenia after intensive chemotherapy with no leukemia relapse or bone marrow blast cells increase. rhG-CSF有促进强烈化疗所致骨髓抑制和粒细胞缺乏的恢复,但未见骨髓原始细胞增多和白血病复发。
The research summary report of active antler resistance the depressed marrow and the immune function influence caused chemotherapy 活性鹿茸抵抗化疗所致骨髓功能抑制及免疫功能影响的研究总结报告
Results The effective rate was 83.3% ( CR 6 cases, PR 9 cases). The main toxicity was depressed marrow. 结果CR6例,PR9例,有效率833%,主要毒性为骨髓抑制。
It was discovered that FLL could significantly promote the rate productive of colony of CFU-S in the depressed marrow, but could not stimulate the proliferation and differentiation of CFU-D in the diffussion chamber. 发现无细胞胎肝液能明显促进受抑制骨髓中多能造血干细胞(CFUS)的集落形成率,但对扩散盒内粒系祖细胞(CFUD)的增殖分化无促进作用。
The results indicated that PNS could partially restore the activity of the depressed bone marrow stem cell proliferation and splenocyte proliferation in mice, and enhance IL-3. IL-6 like substances produced from the splenocytes in these mice. 结果表明PNS可部分恢复CY抑制骨髓细胞增殖能力和脾细胞的活性,且明显提高受抑小鼠IL3,IL6样物质的产生。